Cardiovascular Disease Doctors in St. Louis, MO Accepting Medicare

3 Cardiovascular Disease doctors found. Showing 1 - 3

Ronald Nathan Riner, M.D. Internal Medicine - Cardiovascular Disease Medicare: Not Enrolled in Medicare Practice Location: 9847 Waterbury Drive, St. Louis, MO 63124 Phone: 314-616-8472    Fax: 239-592-5065

Allen D Soffer, MD Internal Medicine - Cardiovascular Disease Medicare: Accepting Medicare Assignments Practice Location: 450 N. New Ballas Rd., Suite 270 West Wing, St. Louis, MO 63141 Phone: 314-991-6969    Fax: 314-997-6969

Leni Abraham, MD Internal Medicine - Cardiovascular Disease Medicare: Accepting Medicare Assignments Practice Location: 3635 Vista Avenue, Desloge Tower, 13th Floor, St. Louis, MO 63110 Phone: 314-268-7992

News Archive

Misperceptions on the chance of seizure freedom after two failed treatments

A common misperception has spread throughout the epilepsy community: It is said that if the first two anti-seizure medications fail, chances of seizure freedom drop to less than 5%.

Preclinical data from Teva's COPAXONE study on multiple sclerosis

Teva Pharmaceutical Industries Ltd. today announced preclinical data demonstrating reparative and neuroprotective effects of treatment with COPAXONE (glatiramer acetate injection) in experimental autoimmune encephalomyelitis (EAE) models.

Study suggests Golgi as potential drug target for Alzheimer's treatment

Alzheimer's disease (AD) progresses inside the brain in a rising storm of cellular chaos as deposits of the toxic protein, amyloid-beta (Aβ), overwhelm neurons. An apparent side effect of accumulating Aβ in neurons is the fragmentation of the Golgi apparatus, the part of the cell involved in packaging and sorting protein cargo including the precursor of Aβ. But is the destruction the Golgi a kind of collateral damage from the Aβ storm or is the loss of Golgi function itself part of the driving force behind Alzheimer's? This was the question for Yanzhuang Wang, Gunjan Joshi, and colleagues at the University of Michigan, Ann Arbor, as they set out to uncover the mechanism damaging the Golgi, using a transgenic mouse and tissue culture models of AD to look at what was going on.

DNA-encoded antibody libraries

During the past several years, breakthroughs in genomic and proteomic technologies have enabled researchers to more clearly identify the biochemical networks that malfunction when a healthy cell becomes malignant.

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