Shere Herrington, | |
1644 Carter St # B, Vidalia, LA 71373-3143 | |
(318) 414-3065 | |
(318) 414-3067 |
Full Name | Shere Herrington |
---|---|
Gender | Female |
Speciality | Case Manager/care Coordinator |
Location | 1644 Carter St # B, Vidalia, Louisiana |
Accepts Medicare Assignments | Does not participate in Medicare Program. She may not accept medicare assignment. |
Identifier | Type | State | Issuer |
---|---|---|---|
1154793859 | NPI | - | NPPES |
Taxonomy | Type | License (State) | Status |
---|---|---|---|
101YM0800X | Counselor - Mental Health | (* (Not Available)) | Secondary |
171M00000X | Case Manager/care Coordinator | (* (Not Available)) | Primary |
Mailing Address | Practice Location Address |
---|---|
Shere Herrington, 1644 Carter St # B, Vidalia, LA 71373-3143 Ph: (318) 414-3065 | Shere Herrington, 1644 Carter St # B, Vidalia, LA 71373-3143 Ph: (318) 414-3065 |
News Archive
USA Today reports that a new polls suggests some Americans believe President Obama's efforts to overhaul the health care system could actually make the system's problems worse.
A new study from The Westmead Institute for Medical Research has identified a genetic variant associated with liver fibrosis (scarring) in chronic hepatitis C patients. This finding is a step toward reducing the number of patients requiring liver transplants.
The small decline was credited mainly to somewhat higher enrollments in Medicare and Medicaid, as well as the Children's Health Insurance Program.
Lung cancer is the leading cause of cancer death throughout the world. Standard treatment methods do not usually result in long-term recovery. In addition to the proliferation of the tumour cells, the growth of blood vessels controls tumors development. The blood vessel growth is controlled by several signalling molecules. Scientists from the Max Planck Institute for Heart and Lung Research in Bad Nauheim and Justus Liebig University Giessen have discovered a molecule that plays a key role in this process. They succeeded in reducing tumour growth in their experiments by blocking the phosphodiesterase PDE4.
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