| Qualitas Dental Group, Pc | |
|
17199 N Laurel Park Dr Suite 200 Livonia MI 48152-2683 | |
| (734) 432-9849 | |
| (734) 953-0412 |
| Full Name | Qualitas Dental Group, Pc |
|---|---|
| Speciality | Dentist - General Practice |
| Location | 17199 N Laurel Park Dr, Livonia, Michigan |
| Authorized Official Name and Position | Daniel K Timmons (PRESIDENT) |
| Authorized Official Contact | 7344329849 |
| Accepts Medicare Insurance | This clinic does not participate in Medicare Program. |
| Mailing Address | Practice Location Address |
|---|---|
| Qualitas Dental Group, Pc 17199 N Laurel Park Dr Suite 200 Livonia MI 48152-2683 Ph: (734) 432-9849 | Qualitas Dental Group, Pc 17199 N Laurel Park Dr Suite 200 Livonia MI 48152-2683 Ph: (734) 432-9849 |
| NPI Number | 1154653319 |
|---|---|
| Provider Enumeration Date | 02/12/2010 |
| Last Update Date | 02/12/2010 |
| Identifier | Type | State | Issuer |
|---|---|---|---|
| 1154653319 | NPI | - | NPPES |
| Taxonomy | Type | License (State) | Status |
|---|---|---|---|
| 1223G0001X | Dentist - General Practice | (Michigan) | Primary |
News Archive
Bacteria provide a well-known playground for scientists and the evolution of these earliest life forms has shed important perspective on potential therapies for some of the most common, deadly diseases. Researchers at Case Western Reserve University School of Medicine have now discovered that, the gas nitric oxide (NO), produced in all cells of the human body for natural purposes, plays a fundamental regulatory role in controlling bacterial function, via a signaling mechanism called S-nitrosylation (SNO), which binds NO to protein molecules.
The U.S. Food and Drug Administration today unveiled a plan containing 25 actions it intends to implement during 2011 to improve the most common path to market for medical devices.
In the central nervous system, nerve cells adhere to the extracellular matrix. Type I collagen is the major class of insoluble fibrous proteins in the extracellular matrix. A previous study has shown that neural stem and progenitor cells, cultured on collagen matrices, are able to expand actively and generate neurons.
A study led by researchers at Boston University School of Medicine provides novel insight into the impact that genes may have on Huntington's disease (HD). The study, published online in PLOS Genetics, identified specific small segments of RNA (called micro RNA or miRNA) encoded in DNA in the human genome that are highly expressed in HD.
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