Dr. Richard I Ecker, M.D. Dermatology Medicare: Not Enrolled in Medicare Practice Location: 706 Ne Evans St, Mcminnville, OR 97128 Phone: 503-472-1405 Fax: 503-434-5950 |
News Archive
A large study has shown that children of mothers who are anxious during pregnancy and in the first few years of the child's life have twice the risk of having hyperactivity symptoms at age 16. This work is being presented for the first time at the ECNP Congress in Copenhagen.
A new long-term look at heart attack care and spending in America since the turn of the century shows more survival, more spending, and more variation between hospitals on both scores.
KineMed, Inc. and CHDI Foundation, Inc. announced today an extension of their collaboration that uses KineMed's translational biomarker platform to characterize and track the pathogenesis of Huntington's disease (HD) and evaluate potentially therapeutic interventions.
Concord Medical Services Holdings Limited, a leading specialty hospital management solution provider and operator of the largest network of radiotherapy and diagnostic imaging centers in China, today announced the signing of a definitive agreement under which it will acquire 19.98% of indirect ownership of The University of Texas MD Anderson Cancer Center Proton Therapy Center (MD Anderson Proton Therapy Center), achieved through acquiring a minority interest in the general partner and facility manager.
The recent discovery of more than a thousand genes known as large intergenic non-coding RNAs (or "lincRNAs") opened up a new approach to understanding the function and organization of the genome. That surprising breakthrough is now made even more compelling with the finding that dozens of these lincRNAs are induced by p53 (the most commonly mutated gene in cancer), suggesting that this class of genes plays a critical role in cell development and regulation. Furthermore, the researchers identify one lincRNA in particular (lincRNA-p21), and demonstrate its critical role in suppressing the reading of many genes across the genome following p53 activation.
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