Dr Robert Moore Oneal, MD | |
501 Onondaga St, Ann Arbor, MI 48104-2606 | |
(734) 769-0238 | |
Not Available |
Full Name | Dr Robert Moore Oneal |
---|---|
Gender | Male |
Speciality | Surgery - Plastic And Reconstructive Surgery |
Location | 501 Onondaga St, Ann Arbor, Michigan |
Accepts Medicare Assignments | Medicare enrolled and may accept medicare through third-party reassignment. May prescribe medicare part D drugs. |
Identifier | Type | State | Issuer |
---|---|---|---|
1851689525 | NPI | - | NPPES |
Taxonomy | Type | License (State) | Status |
---|---|---|---|
2086S0122X | Surgery - Plastic And Reconstructive Surgery | 4301023977 (Michigan) | Primary |
Mailing Address | Practice Location Address |
---|---|
Dr Robert Moore Oneal, MD 501 Onondaga St, Ann Arbor, MI 48104-2606 Ph: (734) 769-0238 | Dr Robert Moore Oneal, MD 501 Onondaga St, Ann Arbor, MI 48104-2606 Ph: (734) 769-0238 |
News Archive
In this post in the Guardian's "Poverty Matters" blog, Prue Clarke, an Africa correspondent, media development specialist and the co-founder and executive director of New Narratives - Africans Reporting Africa, writes, "By not supporting journalists, aid agencies are severely limiting their access to the truth about what is happening in developing countries and, therefore, their ability to make a difference."
Scientists at the Gladstone Institutes have identified networks of genes that play an important role in embryonic-heart development, advancing knowledge of how healthy hearts develop-and offering clues about how to combat a common birth defect known as congenital heart disease.
Two new drug compounds - one of which has already proven useful in a mouse model of multiple sclerosis - appear to be effective in treating endometriosis, a disorder that, like MS, is driven by estrogen and inflammation, scientists report in Science Translational Medicine.
New research supported by the National Institutes of Health delineates how two relatively common variations in a gene called KIF3A are responsible for an impaired skin barrier that allows increased water loss from the skin, promoting the development of atopic dermatitis, commonly known as eczema.
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