Katie R Zechar, MD Hospitalist Medicare: Medicare Enrolled Practice Location: 44428 Woodward Ave Ste 102, Pontiac, MI 48341 Phone: 248-858-3126 Fax: 248-858-6499 |
Dr. Ashvin Karthik Tadakamalla, M.D. Hospitalist Medicare: Accepting Medicare Assignments Practice Location: 44405 Woodward Ave, Pontiac, MI 48341 Phone: 248-858-3234 |
Abdulkader Farran, MD Hospitalist Medicare: Accepting Medicare Assignments Practice Location: 44405 Woodward Ave, Pontiac, MI 48341 Phone: 285-858-3000 |
Dr. Quratulain Durrani, M.D. Hospitalist Medicare: Accepting Medicare Assignments Practice Location: 44405 Woodward Ave, Pontiac, MI 48341 Phone: 248-858-3000 Fax: 248-858-3244 |
News Archive
David Cella, chair of medical social sciences at Northwestern University Feinberg School of Medicine, has received a prestigious international award for his contributions as a pioneer in the field of patient-reported outcome measures and his mission to put the patient's voice in the center of health care.
Racial discrimination may be harmful to your health, according to new research from Rice University sociologists Jenifer Bratter and Bridget Gorman.
Optimer Pharmaceuticals, Inc. today announced that the Committee for Medicinal Products for Human Use (CHMP) of the European Medicines Agency (EMA) has adopted a positive opinion for DIFICLIR (fidaxomicin) tablets for the treatment of adults suffering with a Clostridium difficile infection (CDI), also known as C. difficile-associated diarrhea.
Joint injury can result in irreversible damage of cartilage which, despite treatment and surgery, often eventually leads to osteoarthritis (OA) in later life. New research published in BioMed Central's open access journal Arthritis Research & Therapy demonstrates that short term treatment of damaged cartilage with glucocorticoids can reduce long term degenerative changes and may provide hope for prevention of OA after injury.
Resistance to CD19 CAR T-cell therapy, a type of immunotherapy that yields long-lasting remissions in many patients with B-cell leukemia, can be caused by CD19 splicing alterations, leading to loss of certain parts of the CD19 protein that are recognized by the CAR T cells.
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