| Paul Weisemann Professional Christian Counseling Llc | |
|
138 E Bloomingdale Ave Brandon FL 33511-8101 | |
| (813) 643-2600 | |
| (813) 643-2600 |
| Full Name | Paul Weisemann Professional Christian Counseling Llc |
|---|---|
| Speciality | Counselor - Professional |
| Location | 138 E Bloomingdale Ave, Brandon, Florida |
| Authorized Official Name and Position | Paul M Weisemann (DIRECTOR/THERAPIST) |
| Authorized Official Contact | 8136432600 |
| Accepts Medicare Insurance | This clinic does not participate in Medicare Program. |
| Mailing Address | Practice Location Address |
|---|---|
| Paul Weisemann Professional Christian Counseling Llc 138 E Bloomingdale Ave Brandon FL 33511-8101 Ph: (813) 643-2600 | Paul Weisemann Professional Christian Counseling Llc 138 E Bloomingdale Ave Brandon FL 33511-8101 Ph: (813) 643-2600 |
| NPI Number | 1003095779 |
|---|---|
| Provider Enumeration Date | 10/30/2007 |
| Last Update Date | 05/24/2013 |
| Identifier | Type | State | Issuer |
|---|---|---|---|
| 1003095779 | NPI | - | NPPES |
| Taxonomy | Type | License (State) | Status |
|---|---|---|---|
| 101YP2500X | Counselor - Professional | (* (Not Available)) | Primary |
News Archive
The Committee for Medicinal Products for Human Use (CHMP) of the The European Medicines Agency (EMA) has recommended today to lift the EU-wide suspension of the marketing authorizations of both octagam® and octagam®10% which has been in place since September 2010, subject to certain conditions.
The 2012 South African TB Conference opened Tuesday night in Durban, with the Treatment Action Campaign, Section27, and Oxfam delivering "a memorandum containing five demands to conference organizers shortly before the opening," health-e News Service reports.
A study, performed in mice and utilizing post-mortem samples of brains from patients with Alzheimer's disease, found that a single event of a moderate-to-severe traumatic brain injury (TBI) can disrupt proteins that regulate an enzyme associated with Alzheimer's. The paper, published in The Journal of Neuroscience, identifies the complex mechanisms that result in a rapid and robust post-injury elevation of the enzyme, BACE1, in the brain. These results may lead to the development of a drug treatment that targets this mechanism to slow the progression of Alzheimer's disease.
Most people who carry a genetic mutation for a movement disorder called dystonia will never develop symptoms, a phenomenon that has puzzled scientists since the first genetic mutation was identified in the 1990's.
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