| Scrx-lifetransitions | |
|
222 Saint John St Ste 208 Portland ME 04102-3063 | |
| (207) 891-8618 | |
| Not Available |
| Full Name | Scrx-lifetransitions |
|---|---|
| Speciality | Counselor - Mental Health |
| Location | 222 Saint John St Ste 208, Portland, Maine |
| Authorized Official Name and Position | David Alan Patch (OWNER) |
| Authorized Official Contact | 2078918618 |
| Accepts Medicare Insurance | This clinic does not participate in Medicare Program. |
| Mailing Address | Practice Location Address |
|---|---|
| Scrx-lifetransitions 222 Saint John St Ste 208 Portland ME 04102-3063 Ph: (207) 891-8618 | Scrx-lifetransitions 222 Saint John St Ste 208 Portland ME 04102-3063 Ph: (207) 891-8618 |
| NPI Number | 1043611338 |
|---|---|
| Provider Enumeration Date | 09/06/2014 |
| Last Update Date | 08/16/2023 |
| Certification Date | 08/16/2023 |
| Identifier | Type | State | Issuer |
|---|---|---|---|
| 1043611338 | NPI | - | NPPES |
| Taxonomy | Type | License (State) | Status |
|---|---|---|---|
| 101YM0800X | Counselor - Mental Health | CC-2475 (Maine) | Primary |
News Archive
A new report by University of Southampton academics emphasises the links between poor diet in mothers and ill-health in their children, and calls for women of childbearing age to be made more aware of the importance of good nutrition.
A comprehensive RNA and DNA sequencing platform benefits late-stage and drug-resistant multiple myeloma patients by determining which drugs would work best for them, according to results from a clinical trial published in JCO Precision Oncology in August.
Air quality has been associated with numerous adverse health outcomes from asthma to pre-term birth. Researchers at University of Utah Health found women living along the Wasatch Front - the most populous region in the state of Utah - had a higher risk (16 percent) of miscarriage following short-term exposure to elevated air pollution. The results are available online on December 5 in the journal Fertility and Sterility.
The lack of a gene called LBP-1a in the mouse embryo prevents normal growth of blood vessels in the placenta. This finding suggests that a similar defect in humans could play a role in fetal growth retardation, infant mortality and spontaneous abortion.
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