| Mr Bradley W Jones, CRNA | |
|
830 S Gloster St, Tupelo, MS 38801-4934 | |
| (662) 377-4394 | |
| (662) 377-7045 |
| Full Name | Mr Bradley W Jones |
|---|---|
| Gender | Male |
| Speciality | Certified Registered Nurse Anesthetist (crna) |
| Experience | 24 Years |
| Location | 830 S Gloster St, Tupelo, Mississippi |
| Accepts Medicare Assignments | Yes. He accepts the Medicare-approved amount; you will not be billed for any more than the Medicare deductible and coinsurance. |
| Identifier | Type | State | Issuer |
|---|---|---|---|
| 1013939370 | NPI | - | NPPES |
| 00123799 | Medicaid | MS |
| Taxonomy | Type | License (State) | Status |
|---|---|---|---|
| 367500000X | Nurse Anesthetist, Certified Registered | R798017 (Mississippi) | Primary |
| Group Practice Name | Group PECOS PAC ID | No. of Members |
|---|---|---|
| North Mississippi Medical Center Inc | 9931010600 | 295 |
News Archive
Alzheimer's disease (AD) progresses inside the brain in a rising storm of cellular chaos as deposits of the toxic protein, amyloid-beta (Aβ), overwhelm neurons. An apparent side effect of accumulating Aβ in neurons is the fragmentation of the Golgi apparatus, the part of the cell involved in packaging and sorting protein cargo including the precursor of Aβ. But is the destruction the Golgi a kind of collateral damage from the Aβ storm or is the loss of Golgi function itself part of the driving force behind Alzheimer's? This was the question for Yanzhuang Wang, Gunjan Joshi, and colleagues at the University of Michigan, Ann Arbor, as they set out to uncover the mechanism damaging the Golgi, using a transgenic mouse and tissue culture models of AD to look at what was going on.
Usha Kiran Tarigopula, deputy director in global health at the Bill & Melinda Gates Foundation, writes about the Foundation's partnership with the state of Bihar, India through the Ananya Alliance "aimed at reducing maternal, newborn, and child mortality by 40 percent by 2015," in this "Inpatient Optimists" blog post, which is part of a series called "Global Conversations on Newborn Health in India."
In discrete locations of the adult brain, new neurons are born from stem cells that differentiate, migrate, and integrate into the existing neural network. This process is implicated in normal brain functions such as memory formation and is disrupted in many disease states, including Alzheimer's, schizophrenia, and depression.
A DRUG commonly used to prevent and treat bones losing density and becoming brittle - known as osteoporosis - may also help reduce the risk of breast cancer by one third or more according to new research published in the British Journal of Cancer today (Wednesday).
Acceleron Pharma, Inc., a biopharmaceutical company developing protein therapeutics for cancer and orphan diseases, presented preclinical data highlighting the significant anabolic bone effects in normal and osteopenic mice of RAP-661.
› Verified 4 days ago
| Entity Name | North Mississippi Medical Center Inc |
|---|---|
| Entity Type | Part B Supplier - Clinic/group Practice |
| Entity Identifiers | NPI Number: 1972608347 PECOS PAC ID: 9931010600 Enrollment ID: O20040413000541 |
News Archive
Alzheimer's disease (AD) progresses inside the brain in a rising storm of cellular chaos as deposits of the toxic protein, amyloid-beta (Aβ), overwhelm neurons. An apparent side effect of accumulating Aβ in neurons is the fragmentation of the Golgi apparatus, the part of the cell involved in packaging and sorting protein cargo including the precursor of Aβ. But is the destruction the Golgi a kind of collateral damage from the Aβ storm or is the loss of Golgi function itself part of the driving force behind Alzheimer's? This was the question for Yanzhuang Wang, Gunjan Joshi, and colleagues at the University of Michigan, Ann Arbor, as they set out to uncover the mechanism damaging the Golgi, using a transgenic mouse and tissue culture models of AD to look at what was going on.
Usha Kiran Tarigopula, deputy director in global health at the Bill & Melinda Gates Foundation, writes about the Foundation's partnership with the state of Bihar, India through the Ananya Alliance "aimed at reducing maternal, newborn, and child mortality by 40 percent by 2015," in this "Inpatient Optimists" blog post, which is part of a series called "Global Conversations on Newborn Health in India."
In discrete locations of the adult brain, new neurons are born from stem cells that differentiate, migrate, and integrate into the existing neural network. This process is implicated in normal brain functions such as memory formation and is disrupted in many disease states, including Alzheimer's, schizophrenia, and depression.
A DRUG commonly used to prevent and treat bones losing density and becoming brittle - known as osteoporosis - may also help reduce the risk of breast cancer by one third or more according to new research published in the British Journal of Cancer today (Wednesday).
Acceleron Pharma, Inc., a biopharmaceutical company developing protein therapeutics for cancer and orphan diseases, presented preclinical data highlighting the significant anabolic bone effects in normal and osteopenic mice of RAP-661.
› Verified 4 days ago
| Entity Name | Tupelo Anesthesia Group, Pa |
|---|---|
| Entity Type | Part B Supplier - Clinic/group Practice |
| Entity Identifiers | NPI Number: 1558390443 PECOS PAC ID: 4587566344 Enrollment ID: O20040508000197 |
News Archive
Alzheimer's disease (AD) progresses inside the brain in a rising storm of cellular chaos as deposits of the toxic protein, amyloid-beta (Aβ), overwhelm neurons. An apparent side effect of accumulating Aβ in neurons is the fragmentation of the Golgi apparatus, the part of the cell involved in packaging and sorting protein cargo including the precursor of Aβ. But is the destruction the Golgi a kind of collateral damage from the Aβ storm or is the loss of Golgi function itself part of the driving force behind Alzheimer's? This was the question for Yanzhuang Wang, Gunjan Joshi, and colleagues at the University of Michigan, Ann Arbor, as they set out to uncover the mechanism damaging the Golgi, using a transgenic mouse and tissue culture models of AD to look at what was going on.
Usha Kiran Tarigopula, deputy director in global health at the Bill & Melinda Gates Foundation, writes about the Foundation's partnership with the state of Bihar, India through the Ananya Alliance "aimed at reducing maternal, newborn, and child mortality by 40 percent by 2015," in this "Inpatient Optimists" blog post, which is part of a series called "Global Conversations on Newborn Health in India."
In discrete locations of the adult brain, new neurons are born from stem cells that differentiate, migrate, and integrate into the existing neural network. This process is implicated in normal brain functions such as memory formation and is disrupted in many disease states, including Alzheimer's, schizophrenia, and depression.
A DRUG commonly used to prevent and treat bones losing density and becoming brittle - known as osteoporosis - may also help reduce the risk of breast cancer by one third or more according to new research published in the British Journal of Cancer today (Wednesday).
Acceleron Pharma, Inc., a biopharmaceutical company developing protein therapeutics for cancer and orphan diseases, presented preclinical data highlighting the significant anabolic bone effects in normal and osteopenic mice of RAP-661.
› Verified 4 days ago
| Entity Name | Trident Anesthesia Llc |
|---|---|
| Entity Type | Part B Supplier - Clinic/group Practice |
| Entity Identifiers | NPI Number: 1053713123 PECOS PAC ID: 6901028315 Enrollment ID: O20141119000230 |
News Archive
Alzheimer's disease (AD) progresses inside the brain in a rising storm of cellular chaos as deposits of the toxic protein, amyloid-beta (Aβ), overwhelm neurons. An apparent side effect of accumulating Aβ in neurons is the fragmentation of the Golgi apparatus, the part of the cell involved in packaging and sorting protein cargo including the precursor of Aβ. But is the destruction the Golgi a kind of collateral damage from the Aβ storm or is the loss of Golgi function itself part of the driving force behind Alzheimer's? This was the question for Yanzhuang Wang, Gunjan Joshi, and colleagues at the University of Michigan, Ann Arbor, as they set out to uncover the mechanism damaging the Golgi, using a transgenic mouse and tissue culture models of AD to look at what was going on.
Usha Kiran Tarigopula, deputy director in global health at the Bill & Melinda Gates Foundation, writes about the Foundation's partnership with the state of Bihar, India through the Ananya Alliance "aimed at reducing maternal, newborn, and child mortality by 40 percent by 2015," in this "Inpatient Optimists" blog post, which is part of a series called "Global Conversations on Newborn Health in India."
In discrete locations of the adult brain, new neurons are born from stem cells that differentiate, migrate, and integrate into the existing neural network. This process is implicated in normal brain functions such as memory formation and is disrupted in many disease states, including Alzheimer's, schizophrenia, and depression.
A DRUG commonly used to prevent and treat bones losing density and becoming brittle - known as osteoporosis - may also help reduce the risk of breast cancer by one third or more according to new research published in the British Journal of Cancer today (Wednesday).
Acceleron Pharma, Inc., a biopharmaceutical company developing protein therapeutics for cancer and orphan diseases, presented preclinical data highlighting the significant anabolic bone effects in normal and osteopenic mice of RAP-661.
› Verified 4 days ago
| Entity Name | Dhs Anesthesia Llc |
|---|---|
| Entity Type | Part B Supplier - Clinic/group Practice |
| Entity Identifiers | NPI Number: 1184147431 PECOS PAC ID: 5395017479 Enrollment ID: O20170824001148 |
News Archive
Alzheimer's disease (AD) progresses inside the brain in a rising storm of cellular chaos as deposits of the toxic protein, amyloid-beta (Aβ), overwhelm neurons. An apparent side effect of accumulating Aβ in neurons is the fragmentation of the Golgi apparatus, the part of the cell involved in packaging and sorting protein cargo including the precursor of Aβ. But is the destruction the Golgi a kind of collateral damage from the Aβ storm or is the loss of Golgi function itself part of the driving force behind Alzheimer's? This was the question for Yanzhuang Wang, Gunjan Joshi, and colleagues at the University of Michigan, Ann Arbor, as they set out to uncover the mechanism damaging the Golgi, using a transgenic mouse and tissue culture models of AD to look at what was going on.
Usha Kiran Tarigopula, deputy director in global health at the Bill & Melinda Gates Foundation, writes about the Foundation's partnership with the state of Bihar, India through the Ananya Alliance "aimed at reducing maternal, newborn, and child mortality by 40 percent by 2015," in this "Inpatient Optimists" blog post, which is part of a series called "Global Conversations on Newborn Health in India."
In discrete locations of the adult brain, new neurons are born from stem cells that differentiate, migrate, and integrate into the existing neural network. This process is implicated in normal brain functions such as memory formation and is disrupted in many disease states, including Alzheimer's, schizophrenia, and depression.
A DRUG commonly used to prevent and treat bones losing density and becoming brittle - known as osteoporosis - may also help reduce the risk of breast cancer by one third or more according to new research published in the British Journal of Cancer today (Wednesday).
Acceleron Pharma, Inc., a biopharmaceutical company developing protein therapeutics for cancer and orphan diseases, presented preclinical data highlighting the significant anabolic bone effects in normal and osteopenic mice of RAP-661.
› Verified 4 days ago
| Entity Name | Monroe Health Services Inc |
|---|---|
| Entity Type | Part B Supplier - Hospital Department(s) |
| Entity Identifiers | NPI Number: 1619559309 PECOS PAC ID: 8224376561 Enrollment ID: O20210602001080 |
News Archive
Alzheimer's disease (AD) progresses inside the brain in a rising storm of cellular chaos as deposits of the toxic protein, amyloid-beta (Aβ), overwhelm neurons. An apparent side effect of accumulating Aβ in neurons is the fragmentation of the Golgi apparatus, the part of the cell involved in packaging and sorting protein cargo including the precursor of Aβ. But is the destruction the Golgi a kind of collateral damage from the Aβ storm or is the loss of Golgi function itself part of the driving force behind Alzheimer's? This was the question for Yanzhuang Wang, Gunjan Joshi, and colleagues at the University of Michigan, Ann Arbor, as they set out to uncover the mechanism damaging the Golgi, using a transgenic mouse and tissue culture models of AD to look at what was going on.
Usha Kiran Tarigopula, deputy director in global health at the Bill & Melinda Gates Foundation, writes about the Foundation's partnership with the state of Bihar, India through the Ananya Alliance "aimed at reducing maternal, newborn, and child mortality by 40 percent by 2015," in this "Inpatient Optimists" blog post, which is part of a series called "Global Conversations on Newborn Health in India."
In discrete locations of the adult brain, new neurons are born from stem cells that differentiate, migrate, and integrate into the existing neural network. This process is implicated in normal brain functions such as memory formation and is disrupted in many disease states, including Alzheimer's, schizophrenia, and depression.
A DRUG commonly used to prevent and treat bones losing density and becoming brittle - known as osteoporosis - may also help reduce the risk of breast cancer by one third or more according to new research published in the British Journal of Cancer today (Wednesday).
Acceleron Pharma, Inc., a biopharmaceutical company developing protein therapeutics for cancer and orphan diseases, presented preclinical data highlighting the significant anabolic bone effects in normal and osteopenic mice of RAP-661.
› Verified 4 days ago
| Mailing Address | Practice Location Address |
|---|---|
| Mr Bradley W Jones, CRNA Po Box 3294, Tupelo, MS 38803-3294 Ph: (662) 377-4394 | Mr Bradley W Jones, CRNA 830 S Gloster St, Tupelo, MS 38801-4934 Ph: (662) 377-4394 |
News Archive
Alzheimer's disease (AD) progresses inside the brain in a rising storm of cellular chaos as deposits of the toxic protein, amyloid-beta (Aβ), overwhelm neurons. An apparent side effect of accumulating Aβ in neurons is the fragmentation of the Golgi apparatus, the part of the cell involved in packaging and sorting protein cargo including the precursor of Aβ. But is the destruction the Golgi a kind of collateral damage from the Aβ storm or is the loss of Golgi function itself part of the driving force behind Alzheimer's? This was the question for Yanzhuang Wang, Gunjan Joshi, and colleagues at the University of Michigan, Ann Arbor, as they set out to uncover the mechanism damaging the Golgi, using a transgenic mouse and tissue culture models of AD to look at what was going on.
Usha Kiran Tarigopula, deputy director in global health at the Bill & Melinda Gates Foundation, writes about the Foundation's partnership with the state of Bihar, India through the Ananya Alliance "aimed at reducing maternal, newborn, and child mortality by 40 percent by 2015," in this "Inpatient Optimists" blog post, which is part of a series called "Global Conversations on Newborn Health in India."
In discrete locations of the adult brain, new neurons are born from stem cells that differentiate, migrate, and integrate into the existing neural network. This process is implicated in normal brain functions such as memory formation and is disrupted in many disease states, including Alzheimer's, schizophrenia, and depression.
A DRUG commonly used to prevent and treat bones losing density and becoming brittle - known as osteoporosis - may also help reduce the risk of breast cancer by one third or more according to new research published in the British Journal of Cancer today (Wednesday).
Acceleron Pharma, Inc., a biopharmaceutical company developing protein therapeutics for cancer and orphan diseases, presented preclinical data highlighting the significant anabolic bone effects in normal and osteopenic mice of RAP-661.
› Verified 4 days ago
Lauren Elizabeth Abel, CRNA Nurse Anesthetist - CR Medicare: Accepting Medicare Assignments Practice Location: 830 S Gloster St, Tupelo, MS 38801 Phone: 662-377-4394 Fax: 662-377-7045 | |
Sarah Cotton Ashmore, CRNA Nurse Anesthetist - CR Medicare: Accepting Medicare Assignments Practice Location: 830 South Gloster, Tupelo, MS 38801 Phone: 662-377-4394 Fax: 662-377-7045 | |
Lauren H. Cook, CRNA Nurse Anesthetist - CR Medicare: Accepting Medicare Assignments Practice Location: 830 S Gloster St, Tupelo, MS 38801 Phone: 662-377-4394 Fax: 662-377-7045 | |
Shannon D Barber, CRNA Nurse Anesthetist - CR Medicare: Not Enrolled in Medicare Practice Location: 830 South Gloster, Tupelo, MS 38801 Phone: 662-377-4394 Fax: 662-377-7045 | |
Jennifer M Little, CRNA Nurse Anesthetist - CR Medicare: Accepting Medicare Assignments Practice Location: 830 South Gloster, Tupelo, MS 38801 Phone: 662-377-4394 Fax: 662-377-7045 | |
Donald R Carter, CRNA Nurse Anesthetist - CR Medicare: Accepting Medicare Assignments Practice Location: 830 South Gloster, Tupelo, MS 38801 Phone: 662-377-4394 Fax: 662-377-7045 |