| Central Family Medicine | |
|
720 Central Ave Kansas City KS 66101-3546 | |
| (913) 321-3343 | |
| (913) 321-3348 |
| Full Name | Central Family Medicine |
|---|---|
| Speciality | Internal Medicine |
| Location | 720 Central Ave, Kansas City, Kansas |
| Authorized Official Name and Position | Mark Pederson (MANAGER) |
| Authorized Official Contact | 9133213343 |
| Accepts Medicare Insurance | This clinic does not participate in Medicare Program. |
| Mailing Address | Practice Location Address |
|---|---|
| Central Family Medicine 720 Central Ave Kansas City KS 66101-3546 Ph: (913) 321-3343 | Central Family Medicine 720 Central Ave Kansas City KS 66101-3546 Ph: (913) 321-3343 |
| NPI Number | 1285831305 |
|---|---|
| Provider Enumeration Date | 07/02/2007 |
| Last Update Date | 11/29/2007 |
| Identifier | Type | State | Issuer |
|---|---|---|---|
| 1285831305 | NPI | - | NPPES |
| Taxonomy | Type | License (State) | Status |
|---|---|---|---|
| 207R00000X | Internal Medicine | 04-13172 (Kansas) | Primary |
News Archive
A new study has identified a link between certain genes affected by testosterone and antibody responses to an influenza vaccine. The findings, published in Proceedings of the National Academy of Sciences, suggest that testosterone levels may partially explain why men often have weaker responses to vaccines than women.
Atrium Medical Corporation, pioneering advancements in biological, mechanical and therapeutic solutions for soft tissue reinforcement, is proud to announce that the company was awarded a three-year national contract from Premier Purchasing Partners L.P., the group purchasing organization of Premier, Inc. (Charlotte, NC).
ImaginAb, and Eurogentec S.A. announce a development collaboration to establish a novel system for producing engineered antibody fragments. The two companies will produce ImaginAb's optimized antibody fragments with Eurogentec's high-performance Pichia pastoris expression system.
Oncotarget Volume 11, Issue 11 reported that in this preclinical study, we characterized the binding affinity and selectivity of quizartinib, a small-molecule inhibitor of FLT3, and AC886, the active metabolite of quizartinib, compared with those of other FLT3 inhibitors.
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