Kaedy Page, LPC Counselor - Mental Health Medicare: Not Enrolled in Medicare Practice Location: 900 E 1st St Ste B, Mcpherson, KS 67460 Phone: 620-205-8876 |
Charles Bell Counselor - Addiction (Substance Use Disorder) Medicare: Not Enrolled in Medicare Practice Location: 107 W Marlin, Mcpherson, KS 67460 Phone: 620-241-5550 Fax: 620-241-5554 |
Mrs. Heather Dawn Hartzell Counselor Medicare: Not Enrolled in Medicare Practice Location: 1329 S Walnut St, Mcpherson, KS 67460 Phone: 479-599-8987 |
Darsha Whorton, LPC Counselor - Professional Medicare: Not Enrolled in Medicare Practice Location: 536 Fox Fire, Mcpherson, KS 67460 Phone: 620-245-0608 |
John Swortwood Counselor - Addiction (Substance Use Disorder) Medicare: Not Enrolled in Medicare Practice Location: 107 W Marlin, Mcpherson, KS 67460 Phone: 620-241-5550 Fax: 620-241-5554 |
Benjamin John Nye, LPC Counselor - Professional Medicare: Not Enrolled in Medicare Practice Location: 427 S Fisher St, Mcpherson, KS 67460 Phone: 760-487-8464 |
Kellory Blanchard, LPC-T Counselor - Professional Medicare: Not Enrolled in Medicare Practice Location: 114 N Main St, Mcpherson, KS 67460 Phone: 620-504-2792 |
News Archive
Atherosclerosis is the leading cause of death in the developed world. While a number of risk factors for atherosclerosis have been defined, scientists continue to study other possible risk factors for this disease. Recent epidemiological and experimental studies link infectious agents with the development of inflammatory atherosclerosis. A hallmark of chronic infection with the oral pathogen Porphyromonas gingivalis is the induction of a chronic inflammatory response.
Taro Pharmaceutical Industries Ltd.("Taro" or the "Company") today announced the preliminary results of its modified "Dutch auction" tender offer to repurchase up to $200 million in value of its ordinary shares, which expired at 12:00 midnight, New York City time, on Monday, December 23, 2013.
Both environmental and genetic risk factors contribute to development of lung cancer. Tobacco smoking is the most well-known environmental risk factor associated with lung cancer. A Dartmouth research team led by Yafang Li, PhD, has conducted a study to display that gene-smoking interactions play important roles in the etiology of lung cancer.
When a cell begins to multiply in a dangerously abnormal way, a series of death signals trigger it to self-destruct before it turns cancerous. Now, in research to appear in the August 15 issue of Genes & Development, Rockefeller University scientists have figured out a way in mice to amplify the signals that tell these precancerous cells to die. The trick: Inactivating a protein that normally helps cells to avoid self-destruction.
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