Benjamin Vonfischer, D.O. Emergency Medicine Medicare: Accepting Medicare Assignments Practice Location: 1 Transam Plaza Dr, Suite 360, Oakbrook Terrace, IL 60181 Phone: 630-785-9100 |
Daniel Joseph Sullivan, MD Emergency Medicine Medicare: Not Enrolled in Medicare Practice Location: 1 Transam Plaza Dr, Suite 360, Oakbrook Terrace, IL 60181 Phone: 630-785-9100 Fax: 630-785-9199 |
Dr. Kristopher David Knopp, M.D. Emergency Medicine Medicare: Accepting Medicare Assignments Practice Location: 1 Transam Drive Suite 360, Oakbrook Terrace, IL 60181 Phone: 630-785-9100 |
News Archive
How do you get to Carnegie Hall? New research on the brain's capacity to learn suggests there's more to it than the adage that "practise makes perfect." A music-training study by scientists at the Montreal Neurological Institute and Hospital -The Neuro, at McGill University and colleagues in Germany found evidence to distinguish the parts of the brain that account for individual talent from the parts that are activated through training.
Rather than testing for individual marker genes or proteins, researchers at the University of California, San Diego (UC San Diego) and the Moores UCSD Cancer Center have evidence that groups, or networks, of interactive genes may be more reliable in determining the likelihood that a form of leukemia is fast-moving or slow-growing.
Millennium Laboratories, Inc., a leading provider of therapeutic drug monitoring and education to physicians and staff treating chronic pain, announced today a national agreement with MultiPlan, the nation's leading comprehensive provider of healthcare cost management solutions. As a participating provider in the PHCS and MultiPlan Networks, Millennium Laboratories will assist participating physicians with state-of-the-art therapeutic drug monitoring services and tools.
Researchers suspect that a protein superstructure called amyloid beta is responsible for much of the neural damage of Alzheimer's disease. A new study at the University of California, San Diego, shows that amyloid beta disrupts one of the brain's anti-oxidant proteins and demonstrates a way to protect that protein, and perhaps others, from amyloid's harmful effects.
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