Caitlin Gentilini, Lcsw | |
57 Exchange St Ste 402 Portland ME 04101-5050 | |
(919) 609-1268 | |
Not Available |
Full Name | Caitlin Gentilini, Lcsw |
---|---|
Speciality | Social Worker - Clinical |
Location | 57 Exchange St Ste 402, Portland, Maine |
Authorized Official Name and Position | Caitlin H Gentilini (OWNER OF PRACTICE) |
Authorized Official Contact | 9196091268 |
Accepts Medicare Insurance | This clinic does not participate in Medicare Program. |
Mailing Address | Practice Location Address |
---|---|
Caitlin Gentilini, Lcsw 1 Running Tide Rd Cape Elizabeth ME 04107-2933 Ph: () - | Caitlin Gentilini, Lcsw 57 Exchange St Ste 402 Portland ME 04101-5050 Ph: (919) 609-1268 |
NPI Number | 1558828244 |
---|---|
Provider Enumeration Date | 02/28/2019 |
Last Update Date | 02/28/2019 |
Identifier | Type | State | Issuer |
---|---|---|---|
1558828244 | NPI | - | NPPES |
Taxonomy | Type | License (State) | Status |
---|---|---|---|
1041C0700X | Social Worker - Clinical | (* (Not Available)) | Primary |
News Archive
Beckman Coulter Life Sciences announce that their Aquios CL flow cytometer has been accepted by the World Health Organization (WHO) Prequalification of In Vitro Diagnostics Programme. It can now be used specifically for the immunologic assessment of patients having, or suspected of having, immune deficiency.
A study involving more than 200,000 people worldwide has identified 29 DNA sequence variations in locations across the human genome that influence blood pressure.
The government report says the failure to report medical errors hampers providers' ability to identify and fix preventable problems. Meanwhile, new Medicare data shows hospitals are making little progress in reducing preventable readmissions.
As the demographic shift to an older population continues, a growing number of men and women will be diagnosed with osteoporosis. In addition to existing drug therapies, certain lifestyle and nutritional factors are known to reduce its risk.
Penn Medicine researchers have discovered that hypermethylation - the epigenetic ability to turn down or turn off a bad gene implicated in 10 to 30 percent of patients with Amyotrophic Lateral Sclerosis (ALS) and Frontotemporal Degeneration (FTD) - serves as a protective barrier inhibiting the development of these diseases.
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