Physical Therapists in Pine Bush, NY Accepting Medicare
4 Physical Therapists found. Showing 1 - 4
 Mr. Fritz Gerhard Meier, PT Physical Therapist Medicare: Accepting Medicare Assignments Practice Location: 155 Center Street, Pine Bush, NY 12566 Phone: 845-744-8801 Fax: 845-744-5526 |
 Theresa Caroselli, P.T. Physical Therapist Medicare: Not Enrolled in Medicare Practice Location: 155 Center St., Pine Bush Physical Therapy, Pine Bush, NY 12566 Phone: 845-744-8801 |
Brian Christopher Farrell, P.T. Physical Therapist Medicare: Accepting Medicare Assignments Practice Location: 127 State Route 302, Pine Bush, NY 12566 Phone: 845-744-3669 |
Mrs. Gretchen Meier, PT Physical Therapist Medicare: Medicare Enrolled Practice Location: 155 Center Street, Pine Bush, NY 12566 Phone: 845-744-8801 Fax: 845-744-5526 |
News Archive
Variant of the dopamine receptor gene may be associated with ADHD
A variant of the dopamine receptor gene may be associated with attention-deficit/hyperactivity disorder (ADHD) and with thinner tissue in areas of the brain that handle attention, but also appears associated with better clinical outcomes among individuals with the disorder, according to a report in the August issue of Archives of General Psychiatry , one of the JAMA/Archives journals.
Blocking RNA-silencing protein in the liver prevents obesity and diabetes in mice
Obesity and its related ailments like type 2 diabetes and fatty liver disease pose a major global health burden, but researchers report in Nature Communications that blocking an RNA-silencing protein in the livers of mice keeps the animals from getting fat and diabetic conditions.
Molecular signals make some women prone to miscarriage
Scientists have identified molecular signals that control whether embryos are accepted by the womb, and that appear to function abnormally in women who have suffered repeated miscarriages.
Researchers identify drug using yeast model that reduces levels of toxic protein in brains of AD patients
Using a yeast model of Alzheimer's disease (AD), Whitehead Institute researchers have identified a drug that reduces levels of the toxic protein fragment amyloid-β (Aβ) and prevents at least some of the cellular damage caused when Aβ accumulates in the brains of AD patients.
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