Mr. Timothy Michael Fullmer, MSPT Physical Therapist Medicare: Not Enrolled in Medicare Practice Location: 150 Broad St, Waverly, NY 14892 Phone: 607-687-2495 |
Anne Demott, BS Physical Therapist Medicare: Not Enrolled in Medicare Practice Location: 150 Broad St Ste 1, Waverly, NY 14892 Phone: 607-742-7207 Fax: 607-565-2750 |
Mrs. Patricia Louise Ward, RPT Physical Therapist Medicare: Not Enrolled in Medicare Practice Location: 97 Center St, Waverly, NY 14892 Phone: 607-565-9944 |
Ms. Amy Kathryn Brown, DPT Physical Therapist Medicare: Not Enrolled in Medicare Practice Location: 87 Ellis Creek Rd, Waverly, NY 14892 Phone: 607-972-5158 |
News Archive
Pure Protein, L.L.C., a biopharmaceutical company specializing in diagnostic and therapeutic reagents for use in immunology research and development, has announced that Chief Scientist William Hildebrand, Ph.D., and his team, have developed soluble Class II human leukocyte antigens (HLA). This represents a significant technology breakthrough that can dramatically impact multiple pharmaceutical and medical sectors and has enormous market potential. Autoimmune disease is a key area of interest for HLA research and product development.
Up to half of all prostate cancer cells have a chromosomal rearrangement that results in a new "fusion" gene and formation of its unique protein - but no one has known how that alteration promotes cancer growth. Now, Weill Cornell Medical College researchers have found that in these cancer cells, the 3-D architecture of DNA, wrapped up in a little ball known as a chromatin, is warped in such a way that a switch has been thrown on thousands of genes, turning them on or off to promote abnormal, unchecked growth. Researchers also found that new chromosomal translocations form, further destabilizing the genome.
Targeting only mosquito larvae and not adult insects in insecticide-based dengue control programs could increase mosquito resistance and decrease human immunity to the disease, particularly in urban areas, according to a study published online in the Lancet on Tuesday, Agence France-Presse reports.
Scientists at the Gladstone Institute of Neurological Disease (GIND) have discovered that two main causes of AD amyloid-beta (Aβ) peptides and apolipoprotein E4 (apoE4) impair the growth of new neurons born in adult brains. What is more, they have identified drug treatments that can normalize the development of these cells even in the presence of Aβ or apoE4. The findings are described in two separate papers published in the current issue of Cell Stem Cell.
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