Radiology Doctors in Hobart, IN Accepting Medicare

6 Radiology doctors found. Showing 1 - 6

Erdogan Tufekcioglu, M.D. Radiology - Diagnostic Radiology Medicare: Not Enrolled in Medicare Practice Location: 1500 S Lake Park Ave, Munster Radiology Group, Hobart, IN 46342 Phone: 219-947-6310

Aram Semerdjian, M.D. Radiology - Diagnostic Radiology Medicare: Not Enrolled in Medicare Practice Location: 1500 S Lake Park Ave, Munster Radiology Group, Hobart, IN 46342 Phone: 219-947-6310

Mikhail F Jeha, M.D. Radiology - Diagnostic Radiology Medicare: Accepting Medicare Assignments Practice Location: 1500 S Lake Park Ave, Munster Radiology Group, Hobart, IN 46342 Phone: 219-947-6310

Dr. Monish Merchant, M.D. Radiology - Diagnostic Radiology Medicare: Accepting Medicare Assignments Practice Location: 201 Main St, Hobart, IN 46342 Phone: 219-476-7246    Fax: 219-476-1713

Jaime J. Cebedo, M.D. Radiology - Diagnostic Radiology Medicare: Not Enrolled in Medicare Practice Location: 1500 S Lake Park Ave, Radiology Department, Hobart, IN 46342 Phone: 219-947-6310

Anwar Ahmad, M.D. Radiology - Radiation Oncology Medicare: Medicare Enrolled Practice Location: 1500 S Lake Park Ave, Hobart, IN 46342 Phone: 219-942-5745    Fax: 219-942-8452

News Archive

Prasco commences shipments of generic prescription ZEGERID Capsules

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Anti-anxiety drug reduces autistic features in Jacobsen syndrome mice

About half of children born with Jacobsen syndrome, a rare inherited disease, experience social and behavioral issues consistent with autism spectrum disorders. Researchers at University of California, San Diego School of Medicine and collaborators developed a mouse model of the disease that also exhibits autism-like social behaviors and used it to unravel the molecular mechanism that connects the genetic defects inherited in Jacobsen syndrome to effects on brain function.

Univita earns Health Utilization Management Accreditation from URAC

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Study suggests potential target for treating familial form of ALS

A healthy motor neuron needs to transport its damaged components from the nerve-muscle connection all the way back to the cell body in the spinal cord. If it cannot, the defective components pile up and the cell becomes sick and dies. Researchers at the NIH's National Institute of Neurological Disorders and Stroke have learned how a mutation in the gene for superoxide dismutase 1 (SOD1), which causes ALS, leads cells to accumulate damaged materials.

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